Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. Alternatively, our power to rescue SERT purpose in KI midbrain synaptoneurosomes through the inhibition of FAK suggests elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by greater pFAK localization in five-HT synapses. Our facts https://clayd368spj8.worldblogged.com/profile